Unraveling Alzheimer's: The Effects of Tau on AB Protein Aggregation

Abstract Alzheimer’s is a neurodegenerative disease characterized by the formation of extracellular amyloid-beta (abeta or AB) plaques and intracellular tau plaques. This experiment explores whether the presence of normal tau-441 protein augments AB-42 aggregation. The concentration at which each protein self-aggregated was identified by creating aggregation curves. A control and Read more…

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