2016 Issue
Unraveling Alzheimer's: The Effects of Tau on AB Protein Aggregation
Abstract Alzheimer’s is a neurodegenerative disease characterized by the formation of extracellular amyloid-beta (abeta or AB) plaques and intracellular tau plaques. This experiment explores whether the presence of normal tau-441 protein augments AB-42 aggregation. The concentration at which each protein self-aggregated was identified by creating aggregation curves. A control and Read more…